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1. A study on infant rats showed that administration of MSG lead to five shrunken glands, including the testes. [1] Other studies have shown that growth hormone, thyroid hormones and many other endocrine functions can be assaulted by excitotoxins. NOTE: Some of the guys reading this information have wondered where their testosterone went. It is entirely possible it was assaulted by excitotoxins.
2. One study on mice showed that aspartate, the excitotoxin in diet drinks, gum and aspartame, did the same kind of neuronal damage to the hypothalamus that previous studies on glutamate exhibited. [2] This study mentioned that the research showed that excitotoxins lead to "subsequent obesity, skeletal stunting and reduced mass of pituitaries, ovaries and testes when administered subcutaneously to infant rodents". Yes, the research is clear that excitotoxins can literally damage (and reduce in size) the future development of your child's testes and ovaries.
3. One study on rats showed that female rats given low dose excitotoxins did severe damage to the hypothalamus with resulting nasty effects on multiple hypothalamic functions. [3]
4. Many studies have shown that excitotoxins given to young rodents leads to obesity. And, of course, obesity in humans leads to a host of health issues.
Neurosurgeon Russell Blaylock, in his must-read book
Excitotoxins: The Taste That Kills, documents that just one bowl of soup
or one diet drink will push blood glutamate/aspartate levels past the threshhold that
can damage the hypothalamus. It doesn't take much of these compounds to do damage
and your children are particularly susceptible.
Scientists have been wondering why testosterone levels have been falling for the past several decades. Undoubtedly, excitotoxins have played a signficant role!
Of course, I'm hoping that by writing this, some children out there will get the chance that we never had - to live in a non-toxic environment unsaturated with neurotoxins.
REFERENCES:
1) Neuroendocrinology, 1978, 26(4):220-8, "Late endocrine effects of administering monosodium glutamate to neonatal rats"
2) JOURNAL OF NEURAL TRANSMISSION, 1974, 35(3):207-215, "Glutamate-type hypothalamic-pituitary syndrome in mice treated with aspartate or cysteate in infancy"
3) Neuroendocrinology, 1986, 42(2):158-66, "Developmental and sex-specific effects of low dose neonatal monosodium glutamate administration on mediobasal hypothalamic chemistry"
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