BELLY FAT: You are getting very low carbs. This can increase inflammation. You have very low testosterone which increases insulin levels. This is not a good combination. You are also not getting many calories, which will also lower testosterone very significantly.
I would like to see where you get this idea from?
European journal of applied physiology, 2010 Apr, 108:1125-1131, is that the only study?
I dont see many details on your pagehttp://www.peaktestosterone.com/Atkins_Low_Carb_Diet
I have a hard time figuring out do you think that low carb=high fat?
This is a page I've referenced before because I didnt understand what it was saying and some of the statements dont fly with other things Ive read
example:A Low Carb diet has also been shown to be much more worse for your mood and outlook. Australian scientists placed participants on a Low Fat Diet or Low Carb Diet and, not surprisingly, found that both groups lost an equal amount of weight. However, the low carb group "felt more angry, depressed and confused" than the Low Fat cohort. 
which isnt what this abstract says, as a low carb diet causes euphoria(see below). and a low carb diet is going to cause more weight loss than a low fat diet, as Ive posted before,as each carb holds water. This is why on a low carb diet, you initially lose so much weight, 10-20lbs, so fast,.....it is water weight, but you lose weight and that statement above doesn't say that.
Which is why Id like to see more details about where those statements come from.Low-carb diets, fasting and euphoria: Is there a link between ketosis and gamma-hydroxybutyrate (GHB)?http://www.ncbi.nlm.nih.gov/pubmed/17011713
And you may be ingesting a lot of mercury which can downgrade your T even further.
And an important point: low T can increase your odds for the deadly visceral (belly/stomach) fat. By any chance is your fat accumulating around the gut?
I had petty much only belly fat and I figured that was because fat is hard to remove from the belly(hips/butt in females) due to adrenoreceptors, which give woman the pear shape and men he apple shape. I know androgens, like testosterone levels, can influence adrenoreceptors, but I would tell somehow to fist look at their total body to see where the fat is.
What is witten below is from Lyle Mcdonaldhttp://www.bodyrecomposition.com/
There are two major classes of adrenoreceptors: beta and alpha, which are found all over
the body. This includes the brain, liver, skeletal muscle, fat cells, heart, blood vessels, etc.; you
name it and there are probably adrenoreceptors there.
The main receptors we need to worry about in human fat cells are alpha-2 receptors and
beta-1 and beta-2 receptors, both of which actively bind the catecholamine hormones. When
catecholamines bind to beta-1,2 receptors, they increase cAMP levels, which increases fat
breakdown. Great. However, when the catecholamines bind alpha-2 receptors, they decrease
cAMP levels which decreases fat breakdown. Not great. But it means that catecholamines,
which I told you were fat mobilizers, can actually send both fat mobilizing and anti-fat mobilizing
signals: by binding to either alpha- or beta-receptors.
So why does this matter? Different areas of bodyfat have different distributions of alpha-2
and beta-2 adrenoreceptors. For example, women's lower bodyfat (hips and thighs) have been
found to have 9 times as many alpha-2 receptors as beta-2 receptors. Some research indicates
that men's abdominal fat is similar
, with more alpha-2 than beta-2 receptors. Now you know
part of why its so difficult to reduce these stubborn fat areas; with a greater number of alpha-2
receptors to bind catecholamines, it's that much more difficult to stimulate fat breakdown in
those fat cells.
Other factors affect adrenoreceptor function as well. Androgens and thyroid tend to
increase the sensitivity of beta-2 receptors to the catecholamines.
This may be part of why men
(who have higher androgens and higher thyroid, on average) lose fat more easily. The factors
controlling alpha-2 adrenoreceptor function aren't as well elucidated.
Back to mobilization: summing up
I should note that insulin pretty much always wins the battle over fat cell metabolism.
That is, even in the face of high catecholamine levels, if insulin is elevated, fat mobilization will be
impaired. As it turns out, this generally doesn't happen under normal conditions. Typically when
insulin is high, the catecholamines are low and vice versa (e.g. during exercise, insulin levels drop
as catecholamine levels go up). There are exceptions of course. If you drink a carb drink during
aerobic exercise, for example, the slight increase in insulin will decrease fat mobilization despite
high levels of catecholamines.
Just remember the following: insulin inhibits fat mobilization and the catecholamines
(generally) increase it. Insulin always wins the battle. So when insulin is high and the
catecholamines are low, fat tends to be stored. When insulin is low and the catecholamines are
high, fat will be mobilized. A bit simplistic? Perhaps. But good enough for the time being. The
real take home message is that, from a fat mobilization standpoint, we want low insulin and high
catecholamine levels. Both can be readily accomplished by altering diet (lowering carbohydrates
and calories) and exercise (which increases catecholamines).":