You have seen my other thread, right? I showed 11 studies - and there are dozens more - that show that saturated fat slows down blood flow. So I guess I just find it very weird that all the coconut oil lovers would say, "Well, God has granted miraculous powers to coconut oil and it is the only saturated fat that does not slow down blood flow." To me that's called avoidance.
It's not avoidance, is that it's less important that it slows blood flow after a meal. When you should be most worried about blood flow is at night, when you sleep, when the blood is thickest. You will get some fat to your bloodstream when you're sleeping, from your fat stores. They will be either saturated or unsaturated. If they're unsaturated your body temperature will be lower which will result in thicker blood.
May I suggest a potential problem with this discussion is defining the term ‘saturated fat.’ Fatty acids nomenclature specifies the number of carbon atoms (C8, C10, C12 etc.) and the number of bonds i.e. saturated fats have a single bond, while unsaturated fats (C18:1 for example) consist of at least one double bond. In 1977 then Senator George McGovern introduced The Dietary Goals for the United States and lumped all saturated fats together as unhealthy. We’ve learned a few things since then.
In terms of the human body there are two metabolic pathways separated by the length of the carbon chain; thus medium chain fatty acids (C6 - C12) follow one pathway while long chain fatty acids (C13-C21) follow another. In simple terms long chain fatty acids enter the blood as lipoproteins to transport triglycerides, phospholipids and to facilitate the exchange of cholesterols between the liver and cells. Medium chain fatty acids do none of that. They are routed directly to the liver by the portal vein and transported straight to the cells as energy. The attachment posted previously shows these pathways.
I’m not disputing the possibilities suggested; just that without the specificity of identifying the individual pathways, the interpretation may be suspect. A paper focused on coconut oil would resolve this issue.
Okay, I am going to pose the same challenge to you that I did to Fathomer on the other thread: tell me how we can raise money to study this most basic of things. We get dozens of these kind of issues every week and Big Pharma isn't going to fund this. So how can we make it happen?
Challenge Accepted - It’s unlikely anyone will fund this in the near future, however…
I’ve been on coconut oil forever (metaphorically). If I see improvement with L-Arginine, that would lend credence to your supposition. If I don’t, I may try adding one more supplement with the help of the forum, but if no effect then, I’m probably done. Keep in mind that I’m reasonably functional now, so it may be that at 67 my expectations are unreasonable. On the other hand, it’s possible the experiment has been completed. That is to say if coconut oil is a disaster, shouldn’t I be near non-functional now.
To be perfectly honest – I’m only pursuing this as a test of my circulatory system. If L-Arginine results in improvement, that suggests an all-out approach may be required for heart health, and coconut oil may have long term negative consequences. As stated in my first post - as a trained scientific researcher I am never 100% sure about anything.